April Fools….Practical Clues to the Lyme Disinformation Campaign

Happy birthday to a professional friend who shall remain nameless. I was searching through some of my textbooks for yet more evidence of published research that proves that Lyme disease is a chronic, persisting infection — and possible additional clues for a particular project upon which I am working that pertains to testing inaccuracies, and information of which popular scientific figures are well aware.

Now those of you who know of my illustrious career in research and medico-political sleuthery, are already aware that I have used research including but not limited to these snippets in everything from legislative texts to private letters, legal debates, anti-trust investigations, various campaigns, and one or more documentary films. I have also submitted countless letters to politicians, advocacy groups, patients, attorneys, court officials in multiple countries, and just about anybody who has ever knocked upon my internet “door” asking for help. So none of this information that I post here is new to me. But I would be willing to bet that it is new to many of you. For all others, I defer to any of my books on Lyme disease, but in particular The Baker’s Dozen and the Lunatic Fringe: Has Junk Science Shifted the Lyme Disease Paradigm?; and God Science: The Secret World of Rampant Genetics, Hidden Illness, and Biotech Profiteering; and my latest, The Fourth Monkey: An Untold History of the Lyme Disease Epidemic. (www.allegorypress.com or amazon.com)

Here is some of the dirt on Lyme disease that has not really been told publicly (except in my work); as well as revelations to a paradigm of the denial of epidemic illness that has been active for many decades.

According to Dr. Goodman of the FDA, early antibiotic response to Lyme disease is dicey.     “Most patients with acute tick-borne infectious diseases do not have diagnostic levels of specific antibodies detectable at the time of clinical presentation…”[1]

Transmission of tick-borne pathogens follows several routes:
Horizontal = transfer from tick to host (most effective through saliva or through breaks in the skin)
Tangential = transfer from enzootic cycle to dead-end host (i.e. animal to insect)
Vertical = from one generation to another by reproduction (achieved by transovarial [egg] transmission)
Transstadial = passage from one life stage to another through molt (i.e. larvae to nymph to adult)
Inhalation of aerosolized organisms from dried tick feces (as occurs with Q fever or tularemia [rabbit fever])

The Truth About the Early Antibody Response
Most patients who have acute tick-borne diseases do not display diagnostic levels of specific antibodies that are detectable at the time of clinical presentation, according to Dr. Goodman of the FDA. However he does indicate that “the majority of immunocompetent patients will ultimately develop such antibodies, usually within 2 to 4 weeks of disease onset.” He also states that this is true even where IgM tests are available (immunoglobulin M). But Dr. Goodman also states this: “less than half of patients with early Lyme disease, acute babesiosis, or any of the ehrlichioses (HGA) will have a positive antibody test at the time of presentation.” How about that folks? This means that <50% of patients will be positive for Lyme, and the rest of the patients will be seronegative! Diagnosic testing is felt to be only confirmatory, particularly because of sensitivity and specificity issues, but also because of cross-reactivity between other pathogenic infections.

“…commonly available serodiagnostic tests are seldom sensitive early in acute infection with organisms such as rickettsia, ehrlichia, anaplasma, and spirochetes; most patients are seronegative at the time they present clinically.”

Here is the truth about Lyme disease: “If not promptly suspected or recognized and appropriately treated, several of the acute tick-borne infections can result in progressive and severe illness, often with nervous system involvement or organ failure…fatalities occur even among healthy young adults.”[2]

In a chapter of a textbook that discusses Lyme Borreliosis, Dr. Allen C. Steere, Jenifer Coburn, and Lisa Glickstein of Massachusetts General Hospital, Harvard Medical School, reveal the following, which goes against the mindset that was promoted by Dr. Steere at the October 27-29, 1994 Dearborn Conference that established the surveillance criteria and diagnostic testing standards which have stood in place ever since. At the conference, Dr. Steere promoted the idea that Lyme disease is over diagnosed and over treated.

However, in this texbook the following information can be found: “During the summer of 1976, 24 patients  with EM were identified in the Lyme, Connecticut, area and followed prospectively…Days to weeks later, 75% of these patients developed arthralgias or arthritis, confirming the link between this skin lesion and Lyme arthritis.” Hmm this is interesting because in my book The Fourth Monkey, I show that Dr. Steere and his team identified an unusual mutated spirochete from the EM skin lesion of a Lyme patient who was bitten May 1, 1981 by a tick on Shelter Island, New York.[2]

This odd spirochete was isolated and named Leptospira Inadai Lyme. I go into great detail about the significance of this spirochete, which oddly enough, was not audibly mentioned to the Lyme community. However just a few months later, Shelter Island ticks would again be collected and analyzed for the presence of these very same mutant spirochetes…although this latter fact was never publicly declared before my books. This mutant spirochete was eventually called Leptonema inadai Lyme and it wound up in the same category as other mutated spirochetes including the leptonema found in the Romanian river, and some bulls and turtles, as well as a 1917 Hideo Noguchi Leptonema that seemed to be a mutation between a human genital spirochete and a leptospira from a Leptospirosis infection.

Hmm….Lyme disease caused by a mutant Leptospira / Leptonema? That is the point of my books folks. Borrelia means simply that some pathogen is tick-transmitted. “Burgdorferi” is named after Dr. Willy Burgdorfer…so Borrelia burgdorferi is simply some sort of pathogen that is tick transmitted. But what kind of pathogen?

Dr. Burgdorfer reportedly first identified the Lyme disease spirochete, but according to my research, aspects of that agent were discovered long before the 1981 discovery of Bb, which was helped along by Dr. Allen Steere and Dr. Alan Barbour (NIH). But that is in my books as well. So maybe you might want to get reading. Oh, I forgot to mention the OrbivirusReovirus link to Bb, and also that Dr. Burgdorfer was studying those as early as the 1950s…along with important little pathogens called spiroplasmas, filaria, and other goodies — a few of which have interesting connections to the Bb spirochete. For example, there is a B31 strain of Bb, and there is a B31 bee-flower spiroplasma….

Yes the Borrelia burgdorferi spirochete was magically discovered and named after Dr. Willy Burgdorfer…although nothing else was said audibly about the funny little leptonema or about some of the many other pathogens like reoviruses that are found within the bellies of ticks….but I go into detail about what, how, and why this happened in God Science and also in The Fourth Monkey. You have to read the whole story, as it is quite revealing.

There is evidence to show that the CDC and the military were tracking these mutated spirochetes as early as 1953…leptonema that Dr. Steere’s team said were “intrinsically present” in the Lyme patient’s EM rash — but which they failed to say were directly related to the Lyme patients symptoms or disease, and which failed to be announced with the discovery of the Bb agent. If a leptospira mutant is part of the Bb spirochete, it explains the cross-reactions, the OspC proteins, the low iodine in Lyme patients (leptospira are sensitive to iodine) and a whole bunch of other things my books cover.

Just two years after the first leptonema surfaced in the highly polluted Dimbovitza River in Romania, soon afterward they were also isolated from the bulls and turtles from Illinois, and the turtles in Georgia by the CDC’s laboratory and down river from the Savannah Nuclear Site. Not coincidentally from the evidence I studied, the US military may have been tracking the formation of these mutants from multiple airborne pathogens (likely released as coal-fired, mixed waste, nuclear fuels/steam, or agricultural insecticides that contain important pathogens like phages) as they studied aerobiology, or air pollution by infectious microbes. They even held a conference in 1968 at IIT in Chicago on the topic.

The story continues in the Steere et al. Lyme Borreliosis article… “Moreover some of the patients also developed neurologic or cardiac abnormalities, which showed that Lyme disease was a complex, multisystem illness.” Hmm if Dr. Steere recognized that Lyme is a complex, multisystem illness as early as 1977, it is curious that these same comments were not championed at the Dearborn conference in 1994 which Dr. Steere largely influenced through his recommendations on diagnostic testing. For those of you who are unaware, Dr. Steere was also lead investigator for the upcoming LYMErix vaccine (SmithKline Beecham) which was in development at the time, was awaiting FDA approval, and for which Dr. Steere would be lead investigator running clinical trials. At Dearborn, Dr. Steere recommended the exclusion of the outer surface proteins (Osp) A and B (OspA, OspB), upon which the vaccine was based. If the illness was “over diagnosed and over treated”, then it could not have also been a “complex, multisystem illness” could it?[3]

So which is it? Is Lyme disease a complex, multisystem illness, or is it overdiagnosed? The personal correspondence of Dr. Steere regarding this very topic is discussed in God Science and in The Fourth Monkey. Watch how one position is taken when discussing Lyme to another physician, while the opposite position is taken to affect public policy. There is no explanation for these apparent bipolar positions.

I haven’t even scratched the surface yet, but are you beginning to get the idea that what the public is being told about this illness is a minimalistic description, whereas in the scientific and professional world, quite the opposite is promoted in textbooks which typically run in the hundreds of dollars — outside the reach of the average person who is likely infected with Lyme disease?

Come on guys. Get real. It is time that you read your own materials,
because the patients, despite our various cognitive deficits, still know how to read…..

just sayin’

[1] Goodman Jesse L (FDA), Dennis David T. (CDC), and Sonenshine Daniel E. (Old Dominion U); (editors). Tick-Borne Diseases of Humans. 2005 ASM Press, Washington, DC. pp. 99.
[2] Steere AC, Malawista SE. 1977. Ann Intern Med. 86:685-698.
[3] Goodman Jesse L (FDA), Dennis David T. (CDC), and Sonenshine Daniel E. (Old Dominion U); (editors). Tick-Borne Diseases of Humans. 2005 ASM Press, Washington, DC. assorted pages.

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